Learn / FMK
Every FMK condition
299 diseases, deficiencies, toxicities and drug mechanisms harvested from every FMK deck — the ones sprinkled through a single bullet as much as the ones with their own slide. Pick what you're given and what you have to name, or let it cycle; distractors come from the same category so you have to discriminate (hypoketotic vs. ketotic hypoglycemia, which GSD, which urea-cycle enzyme…). Every answer shows the whole card with the lecture and slide.
Other · FMK 08.4
Biliary atresia Acquired / not inherited
Defect / target: Congenital obliteration of the extrahepatic bile ducts
Mechanism: Absent bile duct patency in the infant blocks excretion of conjugated bilirubin, producing pathologic obstructive neonatal jaundice.
↑ Accumulates: Conjugated bilirubin· ↓ Deficient: Bile flow to gut, stercobilin
Presentation: Pathologic neonatal jaundice (must be distinguished from physiologic jaundice), pale stool, dark urine
Labs: Conjugated hyperbilirubinemia in a newborn
Treatment: Surgery (Kasai portoenterostomy), liver transplant
Learn the mechanism: Heme degradation & bilirubin (jaundice) →
FMK 08.4 Heme Metabolism · slide 25, 27, 29